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The primary mechanism of Axitinib involves the potent inhibition of vascular endothelial growth factor receptors (VEGFRs) and platelet-derived growth factor receptors (PDGFRs). This action makes it a significant player in developing anti-angiogenic therapies for cancer, as it directly targets the pathways responsible for tumor vascularization and growth. Its established efficacy as a tyrosine kinase inhibitor for RCC underscores its importance in treating advanced renal cell carcinoma.

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