The Dual Action of Dihydromyricetin: Combating Cancer Cell Proliferation and ROS Damage
In the fight against cancer, targeting both uncontrolled cell growth and cellular damage is paramount. Dihydromyricetin (DHM), a natural flavonoid, demonstrates a dual action that makes it a compelling compound in cancer research and potential therapy. NINGBO INNO PHARMCHEM CO.,LTD. sheds light on DHM's dual capabilities.
Firstly, DHM is highly effective at inhibiting cancer cell proliferation. It achieves this by interfering with the cell cycle, specifically by inducing arrest at various phases such as G1/S and G2/M. This disruption prevents cancer cells from dividing uncontrollably, a fundamental characteristic of malignancy. The compound's ability to block cell cycle progression makes it a valuable tool for controlling tumor growth.
Secondly, DHM acts as a powerful scavenger of reactive oxygen species (ROS). While ROS are natural byproducts of metabolism, an excess can lead to oxidative stress, damaging DNA and promoting cancer development. DHM's antioxidant properties help neutralize these harmful molecules, thereby protecting cells from damage and potentially inhibiting cancer initiation and progression. This ROS-scavenging ability complements its antiproliferative effects, offering a comprehensive approach to cancer management.
The combination of inhibiting proliferation and mitigating oxidative stress positions DHM as a significant natural compound with broad anti-cancer potential. NINGBO INNO PHARMCHEM CO.,LTD. is dedicated to supplying high-quality DHM for scientific exploration into these critical anti-cancer mechanisms.
Perspectives & Insights
Quantum Pioneer 24
“In the fight against cancer, targeting both uncontrolled cell growth and cellular damage is paramount.”
Bio Explorer X
“Dihydromyricetin (DHM), a natural flavonoid, demonstrates a dual action that makes it a compelling compound in cancer research and potential therapy.”
Nano Catalyst AI
“It achieves this by interfering with the cell cycle, specifically by inducing arrest at various phases such as G1/S and G2/M.”