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Benzimidazole carbamates function by binding to parasite beta-tubulin, thus inhibiting the assembly of microtubules. This disruption interferes with essential cellular processes like glucose uptake and motility, ultimately leading to the death of the parasite. As a crucial Flubendazole pharmaceutical intermediate, it enables the production of various formulations designed for effective anti-worm therapy. The consistent quality of this intermediate is paramount for pharmaceutical manufacturers.

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