GTS-21 Dihydrochloride: Revolutionizing Cognitive Health and Inflammation Management
Discover the power of selective alpha-7 nicotinic acetylcholine receptor agonism for enhanced brain function and reduced inflammation.
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GTS-21 Dihydrochloride
GTS-21 dihydrochloride stands out as a potent selective alpha-7 nicotinic acetylcholine receptor agonist, designed to deliver significant anti-inflammatory and cognitive enhancing benefits. Its mechanism of action targets the nAChR, offering a novel approach to therapeutic interventions.
- Experience enhanced cognitive function with this selective nAChR agonist for improved brain performance.
- Benefit from its anti-inflammatory properties, crucial for managing various inflammatory conditions.
- Explore its potential in treating sepsis through in vivo studies demonstrating improved GI motility and reduced inflammatory markers.
- Understand its role in regulating the JAK2-STAT3 pathway, contributing to its anti-inflammatory effects.
Key Advantages of Utilizing GTS-21
Advanced Receptor Selectivity
Achieve targeted therapeutic outcomes by leveraging the high selectivity of this alpha-7 nicotinic acetylcholine receptor agonist.
Potent Anti-inflammatory Effects
Combat inflammation effectively, as demonstrated by its ability to attenuate pro-inflammatory cytokine production in various research settings.
Cognitive Function Support
Support and potentially enhance cognitive processes, making it a valuable compound for neurological health research.
Key Applications
Cognitive Health Enhancement
Explore the use of this selective nAChR agonist for improving memory and cognitive function, a key area in nootropic research.
Inflammation Management
Investigate its potential as an anti-inflammatory agent, particularly in conditions like sepsis, to reduce systemic inflammation.
Pharmaceutical Research
Utilize this high-purity compound as a vital pharmaceutical intermediate for developing new therapeutic agents.
Neuroscience Studies
Employ GTS-21 for in-depth neuroscience studies, focusing on the function and modulation of nicotinic acetylcholine receptors.